1.
DEFINITIONS
Dementia
is a clinical syndrome that includes hilagnya intellectual function and memory
so that causes dysfunction of daily living (Brocklehurst and Allen, 1987)
According
Lumbantobing SM (1997), dementia is defined as intellectual
deteriorasikapasitas caused by brain disease
2.
Etiology
Causes of dementia has been made a "mnemonics" as
follows:
D: drugs
(pharmaceuticals) for example: sedative, anti-convulsive penenangm,
anti-depressants, and anti-hypertensive
E: emosional (emotional distress, such as
depression)
M: metabolic and endocrine eg: diabetes, kidney
disorders, liver, thyroid, and electrolyte
N: nutrisioral,
eg: lack of vitamin B1, B6, B12 and folic acid
E: eye
and ear (eye and ear dysfunction), for example: cataracts, conductive deafness
T: tumors
and head trauma
I: infection,
Mass: encephalitis, tuberculosis, and neurosyphilis
A: arteriosklerotik
(complications of atherosclerotic disease, Mass infarkmiokard, heart failure,
etc.)
3.
Pathophysiology
Dementia is a clinical syndrome that includes
loss of intellectual function and memory / memory so severe that causes
dysfunction of everyday life. Dementia can be caused by several things al
infection in a long time, the use of excessive drugs, emotional increased,
nutrients lacking in a long time, as well as severe head trauma. Infections
and drugs are toxins in the body that can cause'm a gets into the blood vessels
to circulate throughout the body including the brain, which then damage the
brain cells.
Emotional
overload can also cause dementia, emotional when blood pressure will rise and
when there is no treatment that will either lead to a stroke, a stroke on the
state of the cause disturbances of motor and sensory function or other. While
in a state of poor nutrition needs dala m lam a period of time will cause
metabolic processes that affect the production of red blood cells and cause
arteriosklerotik which can cause a stroke. In red blood cells decreased to
disrupt the supply of O 2 to the tissues of the body including the brain tissue that
can lead to ischemic brain.
Dementia caused by severe trauma, it is the
originator of the damage to brain cells that would lead to smaller brain volume
and occurs degerasi neurons that would lead to disruption of brain function
4.
CLASSIFICATION
Classification of dementia according to R. Budi, 1999:
a. Degenerative Dementia
Known as the Alzheimer
type dementia is a condition include changes in the number, structure and
function of neurons in certain areas of the brain cortex.
Clinical symptoms:
Phase I
Marked with subjective
memory impairment, poor concentration, and visio-spatial disorder. Environment
that used to be such a foreign, hard to find your way home usually take. Clients
may complain of right and left agnosia. Even at this early phase of a
sense of insight (insight) has been disrupted
Phase II
Occurs signs point to a
focal-cortical damage, although not visible pattern typical deficit. Symptoms
caused by parietal lobe dysfunction (eg agnosia, dyspraxia and akalkulia). Symptoms
may occur neuorogi extensor plantar responses include da some facial weakness. Da
delusions hallucinations may occur, although talks may still appear normal
Phase III
Conversation is
interrupted by weight, may be completely lost. Kklien looks continuous
apatik. Many clients do not recognize themselves or people he knew. With
the progress of the disease, patients are often just lay in bed, either
incontinence uri mupun Alvi. Often accompanied by seizures epile [PTIK
grand mal. Severe neurologic symptoms showed severe disruption of motion
step (gait), muscle tone and picture Kluver-Bucy syndrome mengarahpada (s
apathy, ganggua introduction, uncontrollable mouth movements, hypersexuality,
amnesia, and bulimia). The average length of the disease since the
beginning of dianosis until death ranges from 9.3 years (between 8-15 years). Degenerative
disease is a clinical diagnosis in which the diagnosis was made by autopsy or
brain biopsy
b. Multi-infarct dementia
Is second only to Alzheimer's
type. Senagai obtained due to sequelae of cortical stroke or recurrent
subkoirtikal. Typical symptoms that appeared showed a decrease in
multilevel (stepwise), diamana each acute episode showed a decrease in
cognitive state. This situation lasted progressive, frequent examination
by CT scan showed no lesions. With MRI detected more frequently.
c. Dementia in neurology disease
Often occurs in
Parkinson's disease, korea Huntington and hydrocephalus normal pressure. In
a CT scan or MRI found ventricular dilation out of proportion compared to
cortical brain atrophy similar to the symptoms of subcortical dementia also
found symptoms posture and step (gait) and depression
d. Amnestic syndrome and forgetfulness due to aging
The main symptoms are
impaired memory (memory), whereas in dementia there is interference with the
function of new intellectual terjadai usually causes are:
Ø Thiamine deficiency (often due to excessive
alcohol pemakaina)
Ø Lesions in the brain structure of the middle
temporal (due to trauma or anoxia)
Ø Transient global ischemia (cursory) due to
cerebrovascular isufisiensi
5.
Clinical manifestations
Early manifestations:
a.
Forgetful
b.
Tend to misplace items
c.
Repetition of words or
deeds
d.
The ability to speak or
socialize while still good
e.
Amendment when it
continues on further aggravate the disorder, namely:
f.
Patients may not be able
to work
g.
Not being able to
recognize family
h.
Often lost
Psychological symptoms may start to look al:
a.
Depression
b.
Fear
c.
Can not be silent
d.
Apathetic
e.
Paranoid
Often these symptoms complained of by his family some of these
symptoms may aggravate dementia and can often be controlled with medication
6.
Investigations
a. CT Scan / MRI
To ensure a brain hemorrhage, cerebral
infarction single or multiple, location and magnitude of the presence of
metastatic neoplasms
b. EEG (Elektroencefalografi)
c. Examination laboraatorium Leukocytes, blood
sugar
d. Arteriography
e. Puncture lumabal
7.
MEDICAL MANAGEMENT
Drugs used:
a.
Depression can be given
amitriptiline
b.
Anxiety can be given
lorazepam
c.
Agitation, delusions may
be given klorproma z in
d.
Insomnia can diberiakn
prometazim
8.
HEALTH HISTORY
a.
Assessment of the
medical history should include the normal pattern and behavior danperubahan
including information about the time when they occurred
b.
Browse course of the
disease accurately (mendaadak, gradually, gradial)
c.
Then and now includes
the ability to perform daily activities especially tingakh self-care behaviors
like fooding, toileting, bathing, dressing, and level of daily activity and
attention in the past
d.
Past medical history
Ø A history of drug use, alcohol
Ø Emotional disorders eg depression
Ø Metabolic and endocrine diseases
Ø Dysfunction eyes and ears
Ø Nutritional status is less
Ø Tumor and trauma
Ø Infection
Ø Arteriosklerotik
e.
Physical examination
Cognitive function was assessed with now:
Ø Observsi or measurement of orientation to time,
place, and person
Ø Expenditure and memory settings
Ø The use of language
Ø Ability to deal with problems
Ø Mood / affective / emotional level
f.
For portable mental
status examination (MMSE-PPSM (mini mental state examination))
g.
List of
Questions
|
Valuation
|
1.
What is
the date today? (Month, year)
2.
What day
is this?
3.
Is the
name of this place
4.
What is
the client's phone number?(When there is no phone home address)
5.
How old
is Mr / Ms?
6.
when the
father / mother born? (Date, month, year)
7.
What is
the name of our governor?(Headman / sub-district head / district)
8.
What was
the name before this governor?
9.
Who is
the mother's maiden name anada?
10.
3-3
Countdown, ranging from 20?
|
0-2 errors = good
3-4 errors = mild intellectual
disorders
5-7 errors = impaired intellect
being
8-10 errors = severe intellectual
disorders
Ø If the patient is never allowed school kealahan value of
values above +1
Ø If the patient is more ari high school gdiperbolehkan yan
error -1 from above
|
REFERENCES
1.
Smeltzer, SC & Bare, Brunner
and Suddarth's BG textbook of medical - surgical nursing. 8 th Edition. Rather language: Waluyo, A.
New York: EGC; 2000 (the original book was published in 1996)
2.
Price, SA & Wilson,
LM Pathophysiology: Clinical concept of disease processes. 4 th Edition. Rather language: Grace, P.
New York: EGC; 1994 (the original book was published in 1992)
3.
Doengoes, ME, Moorhouse,
MF, Geissler, AC Nursing care plans: Guidelines for planning and
documenting Patient care. Rather language: Kariasa, IM Jakarta: EGC; 1999
(the original book was published in 1993)
4.
Textbook: Geriatrics
(Elderly Health Sciences). Editor: R. Boedhi Darmojo, H. Hadi Martono.
Jakarta: Hall Publishers FKUI; 1999
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